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Cancer of the thyroid is a disease in which malignant (cancerous) cells are found in the tissues of the thyroid gland. The thyroid gland is located at the base of the throat and produces hormones that help the body function normally. Most patients are between 25 and 65 years old, thyroid cancer is more common in women than in men. It is the most common malignancy of the endocrine (hormone) system. There are four main types of thyroid cancer (depending on the type of cell that the cancer developed in); papillary carcinoma, follicular carcinoma, medullary carcinoma and anaplastic carcinoma. Occasionally other types of cancer (lymphoma, sarcoma and carcinosarcoma) can be found in the thyroid gland. Some thyroid cancers are caused by exposure to radiation and some medullary carcinomas are associated with an inherited condition (multiple endocrine neoplasia). However, in the large majority of cases the cause is unknown
Menu: Thyroid Cancer
Information for Patients and the Public
Information for Health Professionals / Researchers
Latest Research Publications
Molecular Biology of Thyroid Cancers
Multiple Endocrine NeoplasiaInformation Patients and the Public (17 links)
- Thyroid Cancer Treatment
National Cancer Institute
PDQ summaries are written and frequently updated by editorial boards of experts Further info. - Thyroid Cancer
Cancer Research UK
CancerHelp information is examined by both expert and lay reviewers. Content is reviewed every 12 to 18 months. Further info. - Thyroid Cancer
Cancer.Net
Content is peer reviewed and Cancer.Net has an Editorial Board of experts and advocates. Content is reviewed annually or as needed. Further info. - Thyroid cancer
Macmillan Cancer Support
Content is developed by a team of information development nurses and content editors, and reviewed by health professionals. Further info. - Thyroid cancer
NHS Choices
NHS Choices information is quality assured by experts and content is reviewed at least every 2 years. Further info. - ThyCa: Thyroid Cancer Survivors' Association, Inc.
Thyroid Cancer Survivors' Association, Inc.
A non-profit organization of thyroid cancer survivors, family members, and health care professionals, founded in 1995. The site includes extensive information about thyroid cancers. - Thyroid Cancer
Nebraska Medical Center
Dr. Bill Lydiatt, a surgical oncologist and thyroid cancer survivor, explains the symptoms, treatment and why it's important to catch thyroid cancer early. - What You Need To Know About Thyroid Cancer
National Cancer Institute - Association for Multiple Endocrine Neoplasia Disorders
AMEND
A UK-based international patient group set up in 2002 to support and inform anyone affected by or interested in multiple endocrine neoplasia disorders and their associated endocrine tumours. - Australian Thyroid Foundation
Australian Thyroid Foundation
A member-based organisation, with membership fees and donations used to support and educate members with the most up-to-date, research, treatment and medications. - British Thyroid Foundation
BTF
A charity, founded in 1991, dedicated to supporting people with thyroid disorders and helping their families and people around them to understand the condition. - Butterfly Thyroid Cancer Trust
Butterfly Thyroid Cancer Trust
A registered charity dedicated solely to the support of patients with Thyroid Cancer, including a telephone helpline. The Website includes information about thyroid cancer, patient journey and details of radioiodine I131 treatment. - Light of Life Foundation
Light of Life Foundation
A non-profit organization founded in 1997to improve the quality of life of thyroid cancer patients through continual education of the lay public and the medical community, and by promoting research and development to improve thyroid cancer care. Includes a Low Iodine Cookbook. - Thyroid Cancer
Childrens' Oncology Group
Includes information about thyroid cancer in children and young adults, with sections on newly diagnosed, in treatment and after treatment. - Thyroid Cancer Alliance
Thyroid Cancer Alliance
An international network of national thyroid cancer patient support organisations working together to provide support and information to those affected by the disease throughout the world. - Thyroid cancer statistics
Cancer Research UK
Statistics for the UK, including incidence, mortality, survival, risk factors and stats related to treatment and symptom relief. - Thyroid Organizations Netherlands
SON - Translate this page
Includes telephone helpline and forum. SON is a collaborative organization with a medical-scientific advisory council.
Information for Health Professionals / Researchers (16 links)
- PubMed search for publications about Thyroid Cancer - Limit search to: [Reviews]
PubMed Central search for free-access publications about Thyroid Cancer
MeSH term: Thyroid Neoplasms
US National Library of Medicine
PubMed has over 22 million citations for biomedical literature from MEDLINE, life science journals, and online books. Constantly updated. - Thyroid Cancer Treatment
National Cancer Institute
PDQ summaries are written and frequently updated by editorial boards of experts Further info. - Thyroid Cancer
Patient UK
PatientUK content is peer reviewed. Content is reviewed by a team led by a Clinical Editor to reflect new or updated guidance and publications. Further info. - Thyroid Cancer
NHS Evidence
Regularly updated and reviewed. Further info. - 2nd World Congress on Thyroid Cancer
World Congress on Thyroid Cancer
A global multi-disciplinary meeting of all specialists involved in the field of Thyroid Cancer. July 10-14th 2013. Toronto, Canada - Anaplastic Thyroid Carcinoma
Medscape
Detailed referenced article by Anastasios Konstantakos, MD. - British Association of Endocrine and Thyroid Surgeons
BAETS
BAETS is the representative body of British Surgeons who have a specialist interest in surgery of the endocrine glands (thyroid, parathyroid and adrenal). - British Thyroid Association
British Thyroid Association
A non-profit making Learned Society of professional clinical specialist doctors and scientists in the United Kingdom who manage patients with thyroid disease and/or are researching into the thyroid and its diseases in humans. - Follicular Thyroid Carcinoma
Medscape
Detailed referenced article by Luigi Santacroce, MD. - Medullary Thyroid Carcinoma
Medscape
Detailed referenced article by Anastasios Konstantakos, MD. Includes overview of MTC and Multiple Endocrine Neoplasia, presentation, diagnosis, workup and treatment. - Papillary Thyroid Carcinoma
Medscape
Detailed referenced article by Luigi Santacroce, MD. - SEER Stat Fact Sheets: Thyroid
SEER, National Cancer Institute
Overview and specific fact sheets on incidence and mortality, survival and stage, lifetime risk, and prevalence. - Thyroid Cancer
http://www.hemonc101.com/
Dr. Tony Talebi discusses treatment of thyroid cancer and "suspicious" thyroid nodules with Dr. Bryan Kim, Director of Thyroid Diseases, University of Miami. - Thyroid Cancer
Oncolex - Oslo University Hospital (Norway) and MD Andersen (USA)
Detailed reference article covering etiology, histology, staging, metastatic patterns, symptoms, differential diagnoses, prognosis, treatment and follow-up. - Thyroid Cancer Forum- UK
Thyroid Cancer Forum- UK
A national organisation that facilitates communication between doctors and scientists working in the thyroid cancer field. It is a free and independent service available to consultants and senior scientists involved in the treatment of these diseases in the UK and Ireland. - Thyroid cancer statistics
Cancer Research UK
Statistics for the UK, including incidence, mortality, survival, risk factors and stats related to treatment and symptom relief.
Latest Research Publications
This list of publications is regularly updated (Source: PubMed).
Association between BRAF V600E mutation and mortality in patients with papillary thyroid cancer.
JAMA. 2013; 309(14):1493-501 [PubMed]
OBJECTIVE: To investigate the relationship between BRAF V600E mutation and PTC-related mortality.
DESIGN, SETTING, AND PARTICIPANTS: Retrospective study of 1849 patients (1411 women and 438 men) with a median age of 46 years (interquartile range, 34-58 years) and an overall median follow-up time of 33 months (interquartile range, 13-67 months) after initial treatment at 13 centers in 7 countries between 1978 and 2011. MAIN OUTCOMES AND MEASURES: Patient deaths specifically caused by PTC.
RESULTS: Overall, mortality was 5.3% (45/845; 95% CI, 3.9%-7.1%) vs 1.1% (11/1004; 95% CI, 0.5%-2.0%) (P < .001) in BRAF V600E-positive vs mutation-negative patients. Deaths per 1000 person-years in the analysis of all PTC were 12.87 (95% CI, 9.61-17.24) vs 2.52 (95% CI, 1.40-4.55) in BRAF V600E-positive vs mutation-negative patients; the hazard ratio (HR) was 2.66 (95% CI, 1.30-5.43) after adjustment for age at diagnosis, sex, and medical center. Deaths per 1000 person-years in the analysis of the conventional variant of PTC were 11.80 (95% CI, 8.39-16.60) vs 2.25 (95% CI, 1.01-5.00) in BRAF V600E-positive vs mutation-negative patients; the adjusted HR was 3.53 (95% CI, 1.25-9.98). When lymph node metastasis, extrathyroidal invasion, and distant metastasis were also included in the model, the association of BRAF V600E with mortality for all PTC was no longer significant (HR, 1.21; 95% CI, 0.53-2.76). A higher BRAF V600E-associated patient mortality was also observed in several clinicopathological subcategories, but statistical significance was lost with adjustment for patient age, sex, and medical center. For example, in patients with lymph node metastasis, the deaths per 1000 person-years were 26.26 (95% CI, 19.18-35.94) vs 5.93 (95% CI, 2.96-11.86) in BRAF V600E-positive vs mutation-negative patients (unadjusted HR, 4.43 [95% CI, 2.06-9.51]; adjusted HR, 1.46 [95% CI, 0.62-3.47]). In patients with distant tumor metastasis, deaths per 1000 person-years were 87.72 (95% CI, 62.68-122.77) vs 32.28 (95% CI, 16.14-64.55) in BRAF V600E-positive vs mutation-negative patients (unadjusted HR, 2.63 [95% CI, 1.21-5.72]; adjusted HR, 0.84 [95% CI, 0.27-2.62]).
CONCLUSIONS AND RELEVANCE: In this retrospective multicenter study, the presence of the BRAF V600E mutation was significantly associated with increased cancer-related mortality among patients with PTC. Because overall mortality in PTC is low and the association was not independent of tumor features, how to use BRAF V600E to manage mortality risk in patients with PTC is unclear. These findings support further investigation of the prognostic and therapeutic implications of BRAF V600E status in PTC.
Biochemical effects of combined action of gamma-irradiation and paclitaxel on anaplastic thyroid cancer cells.
Ukr Biokhim Zh. 2013 Jan-Feb; 85(1):51-61 [PubMed]
The efficacy of patient-dependent practices on exposure rate in patients undergoing iodine-131 ablation.
Health Phys. 2013; 104(5):454-8 [PubMed]
Investigation of DNA damage by the alkaline comet assay in 131I-treated thyroid cancer patients.
Anal Quant Cytol Histol. 2013; 35(1):36-40 [PubMed]
STUDY DESIGN: Peripheral blood samples were collected from papillary thyroid cancer patients who received 131I by oral administration. Blood samples were taken just before the treatment, on the first day of treatment, and 1 week posttreatment. To determine the radiation-induced DNA damage, alkaline comet assay was performed.
RESULTS: It was found that significantly high levels of DNA damage occurred in first day samples when compared to control samples according to tail moment measurements. Also, a decrease in the level of damage was observed in the 1-week samples.
CONCLUSION: Our observations and data confirmed that treatment with 131I for papilloma thyroid cancer can cause DNA damage in circulating lymphocytes, and the comet assay seemed suitable to assess the effect of radioactive iodine for the patients.
A rare neoplasm of the thyroid gland.
N Z Med J. 2013; 126(1369):75-8 [PubMed]
Uptake of ¹³¹I in households of thyroid cancer patients.
Health Phys. 2013; 104(4):434-6 [PubMed]
Preablation 131-I scans with SPECT/CT in postoperative thyroid cancer patients: what is the impact on staging?
J Clin Endocrinol Metab. 2013; 98(3):1163-71 [PubMed]
OBJECTIVE: To determine the contribution of preablation Iodine 131 (131-I) planar with single-photon emission computed tomography/computed tomography (SPECT/CT; diagnostic [Dx] scans) to differentiated thyroid cancer staging.
DESIGN: Prospective sequential series at university clinic.
METHODS: Using American Joint Committee on Cancer (AJCC) tumor, node, metastasis (TNM) staging, seventh edition 320 patients post-total thyroidectomy were initially staged based on clinical and pathology data (pTN) and then restaged after imaging (TNM). The impact of Dx scans with SPECT/CT on N and M scores, and TNM stage, was assessed in younger, age <45 years, n = 138 (43%), and older, age ≥ 45 years, n = 182 (57%) patients, with subgroup analysis for T1a and T1b tumors.
RESULTS: In younger patients Dx scans detected distant metastases in 5 of 138 patients (4%), and nodal metastases in 61 of 138 patients (44%), including unsuspected nodal metastases in 24 of 63 (38%) patients initially assigned pathologic (p) N0 or pNx. In older patients distant metastases were detected in 18 of 182 patients (10%), and nodal metastases in 51 of 182 patients (28%), including unsuspected nodal metastases in 26 of 108 (24%) patients initially assigned pN0 or pNx. Dx scans detected distant metastases in 2 of 49 (4%) T1a, and 3 of 67 (4.5%) T1b patients.
CONCLUSIONS: Dx scans detected regional metastases in 35% of patients, and distant metastases in 8% of patients. Information acquired with Dx scans changed staging in 4% of younger, and 25% of older patients. Preablation scans with SPECT/CT contribute to staging of thyroid cancer. Identification of regional and distant metastases prior to radioiodine therapy has significant potential to alter patient management.
Molecular pathogenesis and mechanisms of thyroid cancer.
Nat Rev Cancer. 2013; 13(3):184-99 [PubMed]
Intrathyroidal ectopic thymic tissue may mimic thyroid cancer: a case report.
J Pediatr Endocrinol Metab. 2012; 25(9-10):997-1000 [PubMed]
Outpatient hemithyroidectomy: safety and feasibility.
B-ENT. 2012; 8(4):279-83 [PubMed]
METHODOLOGY: Between March 2008 and September 2010 we selected 54 patients who met our inclusion criteria for outpatient hemithyroidectomy. The procedure was carried out through a standard cervicotomy under general anaesthesia. No drains were used. We analysed patient outcome based on complications, unplanned admissions and readmissions.
RESULTS: The mean age of the 54 patients was 46 years, and most of them were women (81%). The mean duration of surgery was 64 minutes, and there were no intra-operative complications. After an observation period of at least 3 hours, 44 patients (81.5%) were discharged as planned. Ten patients (18.5%) required admission for urine retention (n = 1), social circumstances (n = 1), persistence of nausea (n = 3), delayed anaesthesia recovery (n = 4) and patient preference (n = 1). All 10 were discharged the next day, and none were readmitted.
CONCLUSIONS: Our study shows that outpatient hemithyroidectomy performed by experienced surgeons in carefully selected patients can be safe and is associated with a low complication rate. However, this series is small and larger studies are needed to confirm the results.
Selumetinib-enhanced radioiodine uptake in advanced thyroid cancer.
N Engl J Med. 2013; 368(7):623-32 [PubMed] Article available free on PMC after 14/08/2013
METHODS: We conducted a study to determine whether the MAPK kinase (MEK) 1 and MEK2 inhibitor selumetinib (AZD6244, ARRY-142886) could reverse refractoriness to radioiodine in patients with metastatic thyroid cancer. After stimulation with thyrotropin alfa, dosimetry with iodine-124 positron-emission tomography (PET) was performed before and 4 weeks after treatment with selumetinib (75 mg twice daily). If the second iodine-124 PET study indicated that a dose of iodine-131 of 2000 cGy or more could be delivered to the metastatic lesion or lesions, therapeutic radioiodine was administered while the patient was receiving selumetinib.
RESULTS: Of 24 patients screened for the study, 20 could be evaluated. The median age was 61 years (range, 44 to 77), and 11 patients were men. Nine patients had tumors with BRAF mutations, and 5 patients had tumors with mutations of NRAS. Selumetinib increased the uptake of iodine-124 in 12 of the 20 patients (4 of 9 patients with BRAF mutations and 5 of 5 patients with NRAS mutations). Eight of these 12 patients reached the dosimetry threshold for radioiodine therapy, including all 5 patients with NRAS mutations. Of the 8 patients treated with radioiodine, 5 had confirmed partial responses and 3 had stable disease; all patients had decreases in serum thyroglobulin levels (mean reduction, 89%). No toxic effects of grade 3 or higher attributable by the investigators to selumetinib were observed. One patient received a diagnosis of myelodysplastic syndrome more than 51 weeks after radioiodine treatment, with progression to acute leukemia.
CONCLUSIONS: Selumetinib produces clinically meaningful increases in iodine uptake and retention in a subgroup of patients with thyroid cancer that is refractory to radioiodine; the effectiveness may be greater in patients with RAS-mutant disease. (Funded by the American Thyroid Association and others; ClinicalTrials.gov number, NCT00970359.).
Cancer after thyroidectomy: a multi-institutional experience with 1,523 patients.
J Am Coll Surg. 2013; 216(4):571-7; discussion 577-9 [PubMed]
STUDY DESIGN: A total of 2,551 patients underwent thyroidectomy at 3 high-volume institutions. Indeterminate/malignant fine-needle aspiration diagnosis was excluded (n = 1,028). Cancer cases were compared among 1,523 patients with Graves' disease (n = 264), nodular goiter (n = 1,095), and toxic nodular goiter (n = 164). Fisher's exact test, chi-square test, Wilcoxon rank sum, Kruskal-Wallis nonparametric t-tests, and multivariable logistic regression were used.
RESULTS: Overall, 238 (15.6%) cancers were recorded: Graves' disease (6.1%), nodular goiter (17.5%), and toxic nodular goiter (18.3%). Cancer rates were significantly different among these groups (p < 0.01) and significantly higher in nodular goiter and toxic nodular goiter vs Graves' disease (p < 0.01); no significant differences in cancer rates were noted among institutions. Overall, 275 patients had thyroiditis (18%). There was a significant association with younger age, male sex, nodular thyroids, and cancer (p < 0.05). Presence of thyroiditis or performance of preoperative fine-needle aspiration was not associated with cancer. Mean cancer size was 1.1 cm (46% >0.5 cm; 39% >1 cm). Most patients underwent total thyroidectomy (80%).
CONCLUSIONS: These data confirm higher than expected incidental thyroid cancer rates (15.6%) in the largest multi-institutional surgical series to date. Nodular thyroids, males, and young patients were more likely to harbor incidental carcinoma. These data support consideration of initial total thyroidectomy as the preferred approach for patients referred to the surgeon with bilateral nodular disease.
Patient with lingual thyroid and squamous cell carcinoma of the tongue base--case report.
Coll Antropol. 2012; 36 Suppl 2:227-9 [PubMed]
Diffuse sclerosing variant of thyroid carcinoma presenting as Hashimoto thyroiditis: a case report.
Coll Antropol. 2012; 36 Suppl 2:219-21 [PubMed]
PAX8-PPARgamma oncogene in follicular thyroid tumors: RT-PCR and immunohistochemical analyses.
Coll Antropol. 2012; 36 Suppl 2:79-82 [PubMed]
Hypocalcaemia after thyroid surgery for differentiated thyroid carcinoma: preliminary study report.
Coll Antropol. 2012; 36 Suppl 2:73-8 [PubMed]
The value of serum thyreoglobulin levels and whole body (I-131) scintigraphy in the follow-up of the thyroid cancer patients after thyroidectomy.
Coll Antropol. 2012; 36 Suppl 2:67-71 [PubMed]
Thyroid metastasis from breast carcinoma resembling medullary thyroid carcinoma.
Coll Antropol. 2012; 36 Suppl 2:63-6 [PubMed]
Skip metastases in papillary thyroid cancer.
Coll Antropol. 2012; 36 Suppl 2:59-62 [PubMed]
Thyroid cancer in Tuzla region of Bosnia and Herzegovina: a 10-year study (1999-2008).
Coll Antropol. 2012; 36 Suppl 2:53-7 [PubMed]
Papillary thyroid microcarcinoma: clinical and pathological study of 321 cases.
Coll Antropol. 2012; 36 Suppl 2:39-45 [PubMed]
Importance of measurement of thyroglobulin and anti-thyroglobulin antibodies in differentiated thyroid cancer.
Coll Antropol. 2012; 36 Suppl 2:33-8 [PubMed]
Thyroglobulin in washout fluid from lymph node fine-needle aspiration biopsy in papillary thyroid cancer: large-scale validation of the cutoff value to determine malignancy and evaluation of discrepant results.
J Clin Endocrinol Metab. 2013; 98(3):1061-8 [PubMed]
OBJECTIVE: Our aims were to validate the cutoff value of FNA-Tg in diagnosing malignant LNs on a large scale and to investigate the influential factors that could result in the discrepancy between the final diagnosis and FNA-Tg.
DESIGN, SETTING, AND PARTICIPANTS: We conducted a retrospective cohort study based on hospital records with 528 cases of FNA-Tg measurement from 419 PTC patients.
MAIN OUTCOME MEASURE: The cutoff value of FNA-Tg was obtained from receiver operating characteristic analysis with final diagnosis. Binary logistic regression analysis was performed to investigate the influential factors.
RESULTS: In the final diagnosis, 190 LNs were malignant, and 338 LNs were benign. The median FNA-Tg was 521.2 (3676.8) ng/mL in malignant LNs, and 0.1 (0.2) ng/mL in benign LNs. The optimal cutoff value of FNA-Tg in distinguishing malignant LNs from benign LNs was 1.0 ng/mL (sensitivity, 93.2%; specificity, 95.9%) in all cases. Combining FNA-Tg and FNA cytology showed superior diagnostic power (sensitivity, 98.4%; specificity, 94.4%) when compared with diagnostic strategy using either FNA cytology or FNA-Tg alone. FNA-Tg, serum TSH, and serum Tg were higher in nonthyroidectomized patients than in thyroidectomized patients (P < .001, respectively). FNA-Tg was correlated with serum TSH and Tg levels (P < .001, respectively), and binary logistic regression analysis showed that serum TSH suppression and serum Tg presence independently affected the diagnosis made by FNA-Tg.
CONCLUSIONS: Our results validated 1.0 ng/mL of FNA-Tg as a cutoff value for diagnosing LN metastasis of PTC and suggested that serum TSH suppression and serum Tg presence should be considered in diagnosing LN malignancy with FNA-Tg in PTC patients.
RTN4IP1 is down-regulated in thyroid cancer and has tumor-suppressive function.
J Clin Endocrinol Metab. 2013; 98(3):E446-54 [PubMed] Article available free on PMC after 01/03/2014
OBJECTIVE: Because the expression and function of this gene is unknown, we sought to characterize its expression in normal, hyperplastic, and benign and malignant thyroid tissue samples and to evaluate its function in cancer cells.
DESIGN: RTN4IP1 expression was analyzed in normal and hyperplastic thyroid tissue and benign and malignant thyroid tissue samples. In 3 thyroid cancer cell lines (TPC1 from a papillary thyroid cancer, FTC133 from a follicular thyroid cancer, XTC1 from a Hürthle cell carcinoma), small interfering RNA knockdown of RTN4IP1 was used to determine its role in regulating the hallmarks of malignant cell phenotype (cellular proliferation, migration, apoptosis, invasion, tumor spheroid formation, anchorage independent growth).
RESULTS: We found RTN4IP1 mRNA expression was significantly down-regulated in follicular and papillary thyroid cancer as compared with normal, hyperplastic, and benign thyroid neoplasms (P < .05). Moreover, RTN4IP1 mRNA expression was significantly lower in larger papillary thyroid cancers (P < .05). Small interfering RNA knockdown of RTN4IP1 expression increased cellular proliferation (2- to 4-fold) in all 3 of the cell lines tested and increased cellular invasion (1.5- to 3-fold) and migration (2- to 7.5-fold), colony formation (3- to 6-fold), and tumor spheroid formation (P < .05) in 2 of the 3 cell lines tested (FTC-133 and XTC1).
CONCLUSIONS: This is the first study to characterize the expression and function of RTN4IP1 in cancer. Our results demonstrate RTN4IP1 is down-regulated in thyroid cancer and is associated with larger papillary thyroid cancer and that it regulates malignant cell phenotype. These findings, taken together, suggest that RTN4IP1 has a tumor-suppressive function and may regulate thyroid cancer progression.
Update on the management of unusual neuroendocrine tumors: pheochromocytoma and paraganglioma, medullary thyroid cancer and adrenocortical carcinoma.
Semin Oncol. 2013; 40(1):120-33 [PubMed]
Pattern of second primary malignancies in thyroid cancer patients.
Niger J Clin Pract. 2013 Jan-Mar; 16(1):96-9 [PubMed]
Can a gene-expression classifier with high negative predictive value solve the indeterminate thyroid fine-needle aspiration dilemma?
Cancer Cytopathol. 2013; 121(3):116-9 [PubMed]
Increased mortality in patients with differentiated thyroid cancer associated with Graves' disease.
J Clin Endocrinol Metab. 2013; 98(3):1014-21 [PubMed]
OBJECTIVE: The aim of this study was to investigate the long-term disease-specific mortality of nonoccult DTCs occurring in patients with GD compared with DTCs in matched euthyroid control patients.
PATIENTS AND DESIGN: The previously described cohorts of nonoccult DTCs occurring in either patients with GD (DTC-GD, n = 21) or matched euthyroid DTC control patients (n = 70) were compared again after a longer follow-up (50-363.6 months; median, 165.6 months) to compare the major clinical endpoints of persistent/recurrent disease and overall survival. Both cohorts were recruited in 1982-1994 at a single institution. All patients had undergone total thyroidectomy and were followed up according to a standardized protocol.
RESULTS: Persistent/recurrent disease was more frequent in DTC-GD patients than in control patients (P = .0119). Disease-specific mortality was also significantly higher in DTC-GD patients (6 of 21, 28.6%) than in euthyroid control patients (2 of 70, 2.9%) (P = .0001). At the last visit, the percentage of disease-free patients was 57.1% (12 of 21) in the DTC-GD group vs 87.1% (61 of 70) in the control group (P = .0025).
CONCLUSIONS: Nonoccult DTCs occurring in patients with GD cause increased disease-specific mortality compared with DTCs in matched euthyroid control patients. These findings emphasize the need for early diagnosis and aggressive treatment of nonoccult DTCs in patients with GD.
DNA copy number variations characterize benign and malignant thyroid tumors.
J Clin Endocrinol Metab. 2013; 98(3):E558-66 [PubMed] Article available free on PMC after 01/03/2014
OBJECTIVE: Our hypothesis was that thyroid tumor subtypes may show characteristic DNA copy number variation (CNV) patterns, which may further improve the preoperative classification.
DESIGN: Our study cohorts included benign follicular adenomas (FAs), classic papillary thyroid carcinomas (PTCs), and follicular variant PTCs (FVPTCs), the three subtypes most commonly associated with inconclusive preoperative cytopathology.
SETTING: Tissue and FNA samples were obtained at an academic tertiary referral center.
PATIENTS: Cases were identified that underwent partial or complete thyroidectomy for malignant or indeterminate thyroid lesions between 2000 and 2008 and had adequate snap-frozen tissue.
INTERVENTIONS: Pairs of tumor tissue and matching normal thyroid tissue-derived DNA were compared using 550K single-nucleotide polymorphism arrays.
MAIN OUTCOME MEASURE: Statistically significant differences in CNV patterns between tumor subtypes were identified.
RESULTS: Segmental amplifications in chromosomes (Ch) 7 and 12 were more common in FAs than in PTCs or FVPTCs. Additionally, a subset of FAs and FVPTCs showed deletions in Ch22. We identified the 5 CNV-associated genes best at discriminating between FAs and PTCs/FVPTCs, which correctly classified 90% of cases. These 5 Ch12 genes were validated by quantitative genomic PCR and gene expression array analyses on the same patient cohort. The 5-gene signature was then successfully validated against an independent test cohort of benign and malignant tumor samples. Finally, we performed a feasibility study on matched FA-derived intraoperative FNA samples and were able to correctly identify FAs harboring the Ch12 amplification signature, whereas FAs without amplification showed a normal Ch12 signature.
CONCLUSIONS: Thyroid tumor subtypes possess characteristic genomic profiles that may further our understanding of structural genetic changes in thyroid tumor subtypes and may lead to the development of new diagnostic biomarkers in FNA samples.
Single-incision robot-assisted transaxillary surgery for early-stage papillary thyroid cancer.
Ann Otol Rhinol Laryngol. 2012; 121(12):811-5 [PubMed]
METHODS: We performed single-incision transaxillary robot-assisted total thyroidectomy with the da Vinci surgical system in 29 patients (5 male, 24 female; median age, 45 years; age range, 19 to 69 years) with papillary thyroid cancer (cT1 NO) using our proposed technique with selected instrumentation. Preoperative ultrasound examination, fine-needle aspiration cytology, and contrast computed tomographic scanning were performed in all subjects. Ultrasound examination was carried out 1 month after surgery to assess the amount of residual thyroid tissue.
RESULTS: No procedure was converted to conventional open surgery. The mean total operation time was 178.51 +/- 24.18 minutes, and the mean console time was 126.10 +/- 14.47 minutes. A progressive reduction of console time was noted (160 minutes for the first procedure versus 102 minutes for the last one). No cases of permanent hypocalcemia, recurrent laryngeal nerve palsy, or postoperative bleeding occurred. The mean amount of postoperative residual thyroid tissue on ultrasound examination was 8.93 +/- 2.15 mm.
CONCLUSIONS: The single-incision transaxillary robot-assisted total thyroidectomy we propose is a feasible and relatively safe procedure in selected patients with early-stage thyroid cancer.
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